Nevus Bungee Jump The unstoppable Kathryn Thomas takes the plunge for a New Zealand Special of RTE's "Rapid" which I directed in 1999. The minicam has been used many times since, but this was groundbreaking at the time, and - if I say so myself - bloody effective. Kathryn was genuinely petrified before the junp, so the deal was: if she did it, I would too. Never again!
Moles Halo Nevi Dr. James L. Campbell Jr., MD discusses Moles Halo Nevi. See more at PLEASE RATE AND COMMENT!!! Halo nevi occur primarily during adolescence. The average age of onset is 15 years. The incidence in the population is estimated to be 1%. Most halo nevi are located on the trunk; they never occur on palms and soles. They may occur as an isolated phenomenon or several nevi may spontaneously develop halos. A pre-existing nevus develops a surrounding rim of hypopigmentation that heralds the gradual disappearance of the nevus over several months. The depigmented halo is symmetric and round or oval with a sharply demarcated border. Skin biopsy shows a junction or compound nevus surrounded by a dense infiltrate of lymphocytes. There are no melanocytes in the halo area. Halo nevi appear to be a host response directed against the nevus cells. Focal atypical nevus cells may be seen though the majority of the pre-existing nevi are benign. The halo usually re-pigments over a few years. Woods light accentuates the halo. Halo nevi also occur in people with vitiligo. A halo may rarely develop around malignant melanoma, but in such instances it is usually not symmetric. People with halo nevi should have a full skin examination to look for vitiligo and also to screen for melanoma. Removal of a halo nevus is unnecessary unless the nevus itself has atypical features. Parental concern over this impressive change is often reason for a conservative excision. In such cases, the mole ...
Nevus High Wire Bungy - Choccie
Histopathology Skin--Cellular Blue Nevus
Moles Melanocytic Nevi Dr. James L. Campbell Jr., MD discusses Moles Melanocytic Nevi. See more at PLEASE RATE AND COMMENT!!! Nevi are benign skin tumors composed of melanocyte-derived nevus cells. Melanocytes originate from neural crest tissue. Melanocytic nevi are classified by age of onset and by the location and arrangement of nevus cells within the skin. Melanocytic nevi are composed of organized clusters of nevus cells arranged at various levels in the skin. Nevi are present on humans and equally common among males and females. Most adults have between 12 and 20 nevi. Larger numbers of nevi may be a familial trait. Nevi are usually asymptomatic though may on occasion be irritated by clothing or external trauma. Nevi present at birth or that appear during infancy are termed congenital nevi. The appearance of newly acquired nevi reaches a peak during adolescence. Fewer nevi are acquired after age 30. Sun exposure appears to be a stimulus for cell growth of nevi as most acquired nevi appear on sun-exposed skin. Existing nevi may increase in size and become more heavily pigmented during puberty or during pregnancy. Nevi appearing after age 30 should be regarded as suspicious as should acquired nevi appearing in sun-protected areas. Evolution of Lesions Acquired nevi first appear as flat, round, uniformly colored papules. During this growth phase, nevi expand laterally while remaining flat and symmetric. Nevi may be slightly darker in color and slightly raised in the center ...
Dermoscopy Made Simple - Reed nevus This video is part of the Dermoscopy Made Simple series of teaching videos in dermoscopy. Reed nevi grow rapidly, are black and dermatoscopically look alarming but they are benign.
Histopathology Skin--Pigmented Spindle Cell Nevus
Embarrassing Illnesses - Becker's Nevus A man visits the doctor to discuss an extremely hairy mark on his back. This series explores the conditions and ailments that leave many of us a little red-faced and aims to de-stigmatise common complaints.
Moles Atypical nevi - Histology Dr. James L. Campbell Jr., MD discusses Moles Atypical nevi - Histology. See more at PLEASE RATE AND COMMENT!!! The nomenclature and histologic criteria for atypical nevi remains controversial though there is general agreement on the following features Architectural disorder with asymmetry Intraepidermal melanocytes extend beyond the main dermal component Subepidermal fibroplasia Lentiginous melanocytic hyperplasia with spindled or epithelioid melanocytes Nevus cell nests are of variable size and bridge adjacent rete ridges Variable degrees of melanocyte atypia.
Moles Junctional Nevi Dr. James L. Campbell Jr., MD discusses Moles Junctional Nevi. See more at PLEASE RATE AND COMMENT!!! Junctional nevi are most commonly found in children. Junction nevi vary in size from 0.1 to 0.6 cm; some are larger. They appear as flat or slightly raised brown to tan macules with uniform pigmentation that may be slightly irregular. The surface is smooth and flat to slightly elevated, and the border is round or oval and symmetric. Most lesions are hairless. Skin markings are preserved on the surface of the nevus. Nests of nevus cells cluster at the dermoepidermal junction. Junction nevi may change into compound nevi after childhood, but they remain as junction nevi on palms, soles, and the genitalia. Junction nevi are rare at birth and generally develop after the age of 2 years. Degeneration into melanoma is rare.
Moles Intradermal Nevi Dr. James L. Campbell Jr., MD discusses Moles Intradermal Nevi. See more at PLEASE RATE AND COMMENT!!! Intradermal nevi are seen mainly after adolescence. Intradermal nevi are elevated, fleshy, and slightly or moderately pigmented papules. Lesions vary in size from a few millimeters to a centimeter. Dermal nevi are brown or black, but may become lighter or flesh-colored with time. Pigmentation may be arranged in flecks. Course, dark, terminal hairs may grow from the nevus. The variety of shapes reflects the evolutionary process in which moles extend downward with age and nevus cells degenerate or become replaced by fat and fibrous tissue. Dome-shaped lesions are the most common. They generally appear on the face and are symmetric, with a smooth surface. They may be white or translucent, with telangiectatic vessels on the surface mimicking basal cell carcinoma. The structure may be warty or polypoid. Pedunculated lesions with a narrow stalk are located on the trunk, neck, axilla, and groin. They may appear as a soft, flabby, wrinkled sack. Elevated nevi are exposed and are prone to trauma from clothing and other stimuli, often causing them to bleed and inflame, influencing some patients to suspect malignancy. White borders may appear, creating a halo nevus. Degeneration into melanoma is very rare, but dermal nevi may resemble nodular melanoma; therefore, knowledge of duration is important. Nests and cords of nevus cells are found within the dermis; they may ...
Moles Compound Nevi Dr. James L. Campbell Jr., MD discusses Moles Compound Nevi. See more at PLEASE RATE AND COMMENT!!! Compound nevi are slightly or markedly raised pigmented papules. Nests of nevus cells are found both at the dermoepidermal junction and within the dermis. Compound nevi can have an irregular border but are symmetric. The surface may be smooth or slightly papillomatous. The center tends to be more heavily pigmented than the periphery. Hair may be present. If a white halo appears at the periphery of the lesion, it is referred to as a halo nevus. Compound nevi tend to increase in thickness and pigmentation in late childhood and adolescence.
Becker's Nevus Dr. James L. Campbell Jr., MD discusses Becker's Nevus. See more at PLEASE RATE AND COMMENT!!! Becker's nevus is not a nevocellular nevus because it lacks nevus cells. The lesion is a developmental anomaly consisting of either a brown macule, a patch of hair, or both. Nonhairy lesions may later develop hair. The lesions appear in adolescent men on the shoulder, submammary area, and upper and lower back. Becker's nevus varies in size and may enlarge to cover the entire upper arm or shoulder. The border is irregular and sharply demarcated. Malignancy has never been reported. Becker nevus syndrome is the presence of an epithelial nevus showing hyperpigmentation, increased hairiness and hamartomatous augmentation of smooth muscle fibers, and other developmental defects such as ipsilateral hypoplasia of breast and skeletal anomalies including scoliosis, spina bifida occulta, or ipsilateral hypoplasia of a limb. The Becker nevus syndrome usually occurs sporadically. Becker's nevus is usually too large to remove by excision. The hair may be shaved. Laser removal of hair and pigmentation is reported.
Mole Spitz Nevus Dr. James L. Campbell Jr., MD discusses Mole Spitz Nevus. See more at PLEASE RATE AND COMMENT!!! Spitz nevus, or benign juvenile melanoma, is most common in children, but does appear in adults. The term melanoma is used because the clinical and histologic appearance is similar to melanoma. It most often occurs on the face, scalp or legs of pre-adolescent children. They are hairless, red to reddish-pink, dome-shaped papules or nodules with a smooth or warty surface; they vary in size from 0.3 to 1.5 cm. The color is caused by increased vascularity, and bleeding sometimes follows trauma. Spitz nevi are usually solitary but may be multiple. They appear suddenly and, contrary to slowly evolving common moles, patients can sometimes date their onset. The Spitz nevus should be removed for microscopic examination. Histologic differentiation from melanoma is sometimes difficult. Skin biopsy reveals overall architectural order with nested spindle-shaped nevus cells and areas with large pleomorphic nevus cells. Such changes would be worrisome for melanoma in an adult. The lesion and its biologic course are benign. Most dermatologists favor complete excision of Spitz nevi to minimize the risk of recurrence and associated pleomorphism.
Moles Speckled Lentiginous Nevus Dr. James L. Campbell Jr., MD discusses Moles Speckled Lentiginous Nevus. See more at PLEASE RATE AND COMMENT!!! Speckled lentiginous nevus was previously referred to as nevus spilus. It is a common hairless, oval or irregularly shaped brown lesion that is dotted with darker brown-to-black spots. They may appear at any age but usually develop before adulthood and follow a benign persistent course. Lesions can appear at birth or in early infancy as light colored caf?au lait macules. Pigmented macules and papules then develop over a period of months to years. Lesions may be very large. It has been suggested that speckled lentiginous nevus is a subtype of congenital melanocytic nevus. The brown area is usually flat, and the black dots may be slightly elevated and contain typical nevus cells. The spots range from 1 to 3 mm in diameter and may be lentigines, junctional, compound, or intraepidermal nevi. The background hyperpigmentation histologically has the features of a lentigo or caf?au lait macule. There is considerable variation in size, ranging from 1 to 20 cm. The anatomic position or time of onset is not related to sun exposure. Transformation into melanoma is very rare. The risk of transformation may be similar to classic congenital nevi of similar size. Routine excision is not necessary. Biopsy suspicious areas. Speckled lentiginous nevus is flat and necessitates excision and closure if the patient desires removal.
Dermoscopy of Atypical Nevi Examples Dr. Thomas P. Habif, MD discusses Dermoscopy of Atypical Nevi Examples. See more at PLEASE RATE AND COMMENT!!! The following four subtypes are distinctive and due to a specific distribution of hypopigmentation or hyperpigmentation throughout the lesion, namely, central, multifocal, or peripheral. 1. Atypical nevus with central hypopigmentation This is a variant of the reticular type with a more or less centrally situated hypopigmented area and a reticular pattern surrounding the periphery. 2. Atypical nevus with central hyperpigmentation: This type is composed of a rim of prominent pigment network lines at the periphery and a central, diffuse, irregularly outlined black hyperpigmentation. This reticular-homogeneous type with central hyperpigmentation is also know as the hypermelanotic nevus. 3. Atypical nevus with multifocal hypo/hyperpigmentation: There are several, small, isolated hypopigmented areas, that leads to an uneven distribution of the pigment network. Patchy distribution of pigmentation, simulates the uneven pigmentation observed in melanoma. Atypical nevi with the reticular pattern and uneven pigmentation are especially prone to over diagnosis as melanoma. 4. Atypical nevus with peripheral hyperpigmentation: There is a reticular pattern with a prominent eccentric peripheral hyperpigmentation. Eccentric peripheral hyperpigmentation is also often found in malignant melanoma. Therefore atypical nevi with eccentric peripheral hyperpigmentation ...
birth mark,Nevus of Ota laser removal Philippines and Asia.wmv Medlite Q-switched frequency doubled Nd:YAG ( wavelength 532 / 1064 ) is the first pulsed laser available for removal of dark and red tattoos , birth marks , freckles, melasma and sun / age spots.
Moles Atypical Nevi Information Dr. James L. Campbell Jr., MD discusses Moles Atypical Nevi Information. See more at PLEASE RATE AND COMMENT!!! Atypical mole syndrome consists of multiple clinically atypical nevi, together with an increased risk of melanoma. It occurs as a familial syndrome and also sporadically. Atypical nevus syndrome was described in 1978 in families whose members possessed large irregular nevi with a variegated color and a palpable dermal component. Several family members had a history of melanoma. These atypical nevi were considered to be a phenotypic marker for family members at increased risk for developing melanoma. Inheritance is thought to be autosomal dominant with variable penetrance. These clinically and histologically distinctive melanocytic nevi occur as solitary lesions, as multiple lesions in sporadic cases, and as multiple lesions in the familial syndrome. In all three settings, atypical nevi are considered to be precursors for melanoma, most often the superficial spreading type. More than 90% of patients with familial melanoma have atypical nevi.
Mole Recurrent Previously Excised Nevi Dr. James L. Campbell Jr., MD discusses Mole Recurrent Previously Excised Nevi. See more at PLEASE RATE AND COMMENT!!! Weeks to months after incomplete removal of a nevus, brown macular pigmentation may appear in the scar. Some nevus cells remain with shave excision and partial repigmentation is possible. Randomly distributed pigmentation along with scar can be quite suspicious for melanoma. Residual pigmentation may be removed with electrocautery or cryosurgery. An unusual histologic picture resembling melanoma (pseudomelanoma) may follow partial removal of a nevus. If the repigmented area is excised, the pathologist should always be notified that the submitted tissue was acquired from a previously treated area. Histologically, the melanocytes appear atypical but are confined to the epidermis, and there is no lateral spread of individual melanocytes. The history of previous biopsy and a review of the original specimen are critical to the correct diagnosis.
Nevus Bungy SEOptimiser takes on the 134 metre "Nevus Bungy" in Queenstown, New Zealand...and survives! Job's a good 'un!
Melanoma en nevus
One Day at A Time by Nevus Norr When you first put the bottle down you will feel like the hounds of hell have surrounded you but if you hold your ground you'll be fine just take it One Day at a Time Don't look ahead just focus on the now today's the day that counts don't turn around and take it One Day at a Time (Chorus) One Day at a Time -- that's how we all do it One Day at a Time -- that's how we all get through it (Repeat All) Sure there'll come a time you'll want a drop or spot of something like a vodka Stoli's nice, best on ice -- or some rotgut from Kamchatka Oh, you like your brandy, isn't that fine and dandy And here we got a red wine sport, and another who like his drams of port You'll Need a higher, You'll need a Higher, Higher Power To bail you out Hennesey, Courvoisier, Millier Lite will make my day Whiskey's good and bourbon, in the sticks or going urban But it's all a lie -- and it's do or die in a DT fit You'll need a higher power You'll need a higher power Repeat all And the promises The promises will come true Except maybe for that tricky bit About economic insecurity But you WILL be able to handle situation's That used to mess up yo head And you won't need a drink You'll make a better choice instead Take a moment, breathe and think -- And remember when -- You first put the bottle down and it felt like the hounds of hell were surrounding you ... etc. When you start romancing it
Nevus Bungy 134m of adrenaline!! i packed myself when i got there but never thought i would be like that haha i was keen before hand then holy *** nah!! nervs came out
Moles Atypical nevi - Incidence Dr. James L. Campbell Jr., MD discusses Moles Atypical nevi - Incidence. See more at PLEASE RATE AND COMMENT!!! Atypical nevi are common, with a prevalence estimated at 5-20%. While solitary atypical nevi are common, the familial syndrome is uncommon. The incidence of sporadic (non-familial) occurrence of multiple atypical nevi is unknown. Males and females are equally affected. Atypical nevi are not present at birth and begin to appear during early childhood. The characteristic features of atypical moles are present at the time of puberty. Unlike common acquired melanocytic nevi, which stop appearing after age 30, atypical nevi continue to appear well into adulthood. While sun exposure does appear to favor the appearance of atypical nevi, lesions develop in both sun-exposed and sun-protected areas. The number of nevi varies in patients with multiple atypical nevi. Most affected people have more than 50 melanocytic nevi, some of which are atypical in appearance. There is striking heterogeneity from one nevus to another.
Histopathology Skin--Compound Clarks nevus (dysplastic nevus Histopathology Skin--Compound Clarks nevus (dysplastic nevus)
Moles Birthmark Congenital Nevi Dr. James L. Campbell Jr., MD discusses Moles Birthmark Congenital Nevi. See more at PLEASE RATE AND COMMENT!!! Congenital nevi are benign skin tumors composed of melanocyte-derived nevus cells, present at birth, or appearing by age 2. They are considered to be a type of birthmark, of which there are many variants. Roughly 1% of newborns have at least one melanocytic nevus. They may increase in size and become more heavily pigmented during puberty. Congenital melanocytic nevi are usually dark brown and raised, with an irregular verrucous surface. Most have increased course terminal hairs. Congenital melanocytic nevi vary greatly in size. Depending on their location, large lesions may be disfiguring. The largest lesions are referred to as giant hairy nevi. Giant congenital nevi on the trunk are referred to as bathing trunk nevi. Congenital nevi are usually compound nevi with nested nevus cells at the junction and also in the dermis. Nevus cells may extend into fat and invest adnexal structures and blood vessels. The vast majority of congenital melanocytic nevi are benign and follow a life-long course of maturation. Nevi which deviate from this pattern are suspicious and biopsy is warranted. The risk of malignant degeneration occurring in congenital melanocytic nevi is controversial. In general, the risk of malignant change is increased in giant congenital melanocytic nevi with diameters greater than 20 cm with a lifetime risk of 5-8%. The lifetime risk of ...
Moles Atypical nevi - Epidemiology Dr. James L. Campbell Jr., MD discusses Moles Atypical nevi - Epidemiology. See more at PLEASE RATE AND COMMENT!!! Atypical nevi are best regarded as part of a continuum between benign and malignant melanocytic neoplasms. The likelihood of an individual atypical nevus subsequently developing into melanoma cannot be estimated, although the risk of an individual [with atypical nevi can be estimated. The lifetime risk of melanoma for the population born in the 2000 is estimated at 1.3%. The lifetime risk of melanoma for persons with atypical nevi, but without family history of melanoma, has been estimated at 6%. This risk increases to 15% in patients with atypical nevi and a family history of melanoma. Persons with the familial atypical nevus syndrome have a 150-fold greater risk of developing melanoma by age 70 than the general population. This risk increases to a 500-fold greater risk if the patient has already had a melanoma. Thus the lifetime risk approaches 100% by age 70 in patients with the familial atypical nevus syndrome.
Histopathology Skin--Lentiginous junctional nevus, hypermela Histopathology Skin--Lentiginous junctional nevus, hypermelanotic
Nevus Sebaceus Information Dr. Shane Chapman, MD discusses Nevus Sebaceus Information. See more at PLEASE RATE AND COMMENT!!! Nevus sebaceus is a distinctive congenital lesion of the scalp composed of skin and appendageal components. Nearly all nevus sebaceus lesions are present at birth or appear in early childhood. Lesions change clinically and histologically with age. Not all lesions may be noticed at birth. At puberty, these nevi tend to enlarge, and may be noticed for the first time.
Histopathology Skin--Compound nevus with halo reaction
Dermoscopy Made Simple - Spitz nevi This video looks at Spitz nevi. It is part of the Dermoscopy Made Simple series. See
Nevus Reconstruction Surgery of an infant's ear This is the story of our daughter's Nevus removal around her left ear as an infant. Hopefully this will help to ease the pain of any other parents faced with a similar issue. We as the parents were very pleased with Dr. Bauer and his team!
Nevus High Wire Bungy - Fritter
Nevus Sebaceus Treatment Dr. Shane Chapman, MD discusses Nevus Sebaceus Treatment. See more at PLEASE RATE AND COMMENT!!! Excision of the entire lesion is recommended. Nevus sebaceus has a tendency to develop basal cell carcinoma within the nevus after puberty. This is best performed just before puberty, when the lesion is still small and the patient is old enough to understand and tolerate the procedure. A larger lesion may be excised in stages without any concern of inducing malignant change.
Nevus High Wire Bungy - SnookaT
Mole Blue Nevus Dr. James L. Campbell Jr., MD discusses Mole Blue Nevus. See more at PLEASE RATE AND COMMENT!!! The blue nevus is a solitary, slightly elevated, round, regular nevus, usually less than 0.5 cm. Coloration is attributed to intensely pigmented melanocytes located in the mid to lower dermis. The brown pigment absorbs the longer wavelengths of light and scatters blue light. This is called the Tyndall effect. The blue nevus appears in childhood and is most common on the extremities, head and neck, buttock and dorsum of the hands. A rare variant, the cellular blue nevus, is larger (usually greater than 1 cm) and nodular and is frequently located on the buttock. Melanomas are reported arising in association with a common or cellular blue nevus and arising de novo and resembling cellular blue nevi. Blue nevi may be removed for cosmetic purposes.
Nevus Sebaceus Appearance Dr. Shane Chapman, MD discusses Nevus Sebaceus Appearance. See more at PLEASE RATE AND COMMENT!!! Nevus sebaceus is usually a solitary, yellowish to flesh-colored plaque and occurs most commonly on the scalp, forehead, or postauricular areas. It tends to be linear or oval shaped, measuring 1 to 3 cm in diameter. The lesion evolves in three stages corresponding to sebaceous gland maturation through childhood, puberty, and adulthood. In the first few months of life, the sebaceous glands are well developed as a result of maternal hormonal stimulation, although surrounding hair structures are incompletely differentiated. In childhood, the lesion is a barely raised, pink to tan, velvety surfaced hairless plaque and is asymptomatic. The sebaceous glands are small in size and number; incompletely developed hair structures may be seen. With puberty, hormonal influences bring about diagnostic changes. Sebaceous glands mature and increase in size and density. Hair structures remain undifferentiated, and papillomatous epidermal hyperplasia develops. Ectopic apocrine glands may also be found deep within the underlying dermis. The plaque tends to thicken, becomes larger and more verrucous, and has a yellow-white and pink speckled appearance. Lesions at this stage are easily traumatized and may be tender. The third stage of evolution occurs during adulthood. Appendageal tumors may develop within nevus sebaceus. Each such tumor has its own histologic pattern. The most ...
Dr. Samimi: Treatment of a Giant Nevus A chid with a igant nevus covering most of his back is treated by Dr. Samimi with tissue expansion. This news report aired on local TV in Tucson, Arizona and on other US TV stations.
Dr. Samimi: Treatment of Giant Nevus of Nose A boy born with a giant nevus of the nose undergoes treatment by Dr. Samimi for removing it and reconstructing the nose. This news report was aired on TV stations throughout the United States.
Q switch YouTube Professional Nd yag laser treatment of nevus of Ota INDICATIONS Dermis pigmentation, such as lentigines, blue or black tattoo, eyebrow color. Epidermis pigmentation, such as freckle, senior fleck and yellow, red, brown tattoo, lip color, Tattoo removal. Principle: Therapy of epidermis and dermis pigmentation---Utilizing the explosive effect of the Nd Yag laser, the laser permeate the epidermis into the dermis which includes amount of pigments mass. Since the laser pulses in nanosecond but with super high energy, the shot pigment mass swell quickly and breaks into small pieces, which will be eliminated through metabolic system Therapy of the dilatation of capillary vessel---Utilizing the hear effect of the Nd: Yag laser, the laser is absorbed by the hemachrome in the capillary vessel, then the capillary vessel become blocked and disappear.
Excision of a Moderate/Severe Dysplastic Nevus (DPN) 45 YO woman, with a history of Malignant Melanoma, has a Moderate/Severe DPN of her right shin excised by Dr Joseph Kamelgard.
Dermoscopy of Atypical Nevi Dr. Thomas P. Habif, MD discusses Dermoscopy of Atypical Nevi. See more at PLEASE RATE AND COMMENT!!! There are several variations of Clark nevi. One must understand and recognize these variations in order to differentiate these lesions from melanoma. There are limitation in the ability of dermoscopy and physical examination to differentiate between Clark nevi and melanoma in situ or early invasive melanoma. A paper entitled Dermoscopic Classification of Atypical Melanocytic Nevi (Clark Nevi) was published in the Archives of dermatology in 2001. The classification is easily applicable because only 3 dermoscopic structural features (reticular, globular, homogeneous) and 2 descriptors of pigmentation have to be identified. The homogeneous pigmentation is either dark (hyperpigmentation) or light (hypopigmenation). Pigmentation can appear in different distribution patterns. Classification is therefore by predominate structural component and distribution of pigmentation. The three dermoscopic structural types are reticular, globular, and homogeneous. Two structural components may be present in a single lesion. This results in 6 dermoscopic subtypes reticular, globular, homogeneous, reticular-globular, reticular-homogeneous, and globular-homogeneous. The most common type of atypical nevus is the reticular type, followed by the reticular-homogeneous and globular-homogeneous types. No more that three structural components are present in benign atypical nevi. In ...